Congestive Cardiac Failure
Comprehensive Nursing Notes
Congestive Heart Failure (CHF) is a complex clinical syndrome resulting from structural or functional impairment of ventricular filling or ejection of blood. This leads to the hallmark symptoms of dyspnea, fatigue, and fluid retention. As a nursing student, understanding CHF is crucial because it’s one of the most common chronic conditions you’ll encounter in clinical practice.
Quick Facts
- CHF affects approximately 6.2 million adults in the United States
- The 5-year mortality rate after diagnosis is about 50%
- CHF is the leading cause of hospitalization in patients over 65 years old
- Annual healthcare costs exceed $30 billion in the US alone
Congestive heart failure (CHF) represents a progressive disorder in which the heart’s ability to pump blood is compromised. The underlying pathophysiology involves complex mechanisms that attempt to compensate for decreased cardiac output but ultimately worsen the condition.
Heart Failure Pathophysiology Overview
Primary Mechanisms
Heart failure begins with damage to the cardiac muscle that impairs its ability to contract (systolic dysfunction) or relax (diastolic dysfunction). This leads to:
- Decreased cardiac output – Reduced blood flow to organs and tissues
- Venous congestion – Fluid buildup in tissues and organs
- Neurohormonal activation – Body’s attempt to compensate for reduced cardiac output
Compensatory Mechanisms
The body initiates several compensatory mechanisms that initially help maintain cardiac output but eventually become maladaptive:
- Increased heart rate – To maintain cardiac output
- Increased contractility – To enhance stroke volume
- Ventricular hypertrophy – Muscle enlargement to enhance pumping
- Fluid retention – To increase preload
The Frank-Starling mechanism initially helps in heart failure by increasing stroke volume in response to increased preload. However, as the heart continues to fail, this mechanism becomes ineffective as the myocardium stretches beyond its optimal length-tension relationship, leading to worsening cardiac output despite increased filling pressures.
Neurohormonal Activation
Heart failure triggers activation of several neurohormonal systems:
Sympathetic Nervous System
Activates in response to decreased cardiac output, causing:
- Increased heart rate
- Enhanced contractility
- Peripheral vasoconstriction
- Increased myocardial oxygen demand
Renin-Angiotensin-Aldosterone System (RAAS)
Activates in response to decreased renal perfusion, causing:
- Fluid retention (sodium and water)
- Vasoconstriction
- Increased afterload
- Ventricular remodeling
The Vicious Cycle of Heart Failure
Initially compensatory mechanisms become maladaptive over time, leading to a vicious cycle:
- Decreased cardiac output activates compensatory mechanisms
- Compensatory mechanisms increase cardiac workload
- Increased workload worsens myocardial function
- Further decrease in cardiac output
- Progressive cardiac remodeling and ventricular dilation
Heart failure is classified in several ways, based on the affected side, ejection fraction, time course, and functional capacity. Understanding these classifications is essential for appropriate nursing care.
Anatomical Classification
Feature | Left-Sided Heart Failure | Right-Sided Heart Failure | Biventricular Failure |
---|---|---|---|
Primary Issue | Left ventricle cannot pump efficiently | Right ventricle cannot pump efficiently | Both ventricles fail |
Congestion Location | Pulmonary circulation | Systemic circulation | Both circulations |
Key Symptoms | Dyspnea, orthopnea, pulmonary edema, fatigue | Peripheral edema, ascites, hepatomegaly, JVD | All symptoms combined |
Auscultation Findings | Pulmonary crackles, S3 heart sound | Clear lungs, hepatojugular reflux | Combined findings |
Common Causes | Coronary artery disease, hypertension, aortic valve disease | Left-sided heart failure, pulmonary hypertension, COPD | Advanced heart disease |
Functional Classification
The New York Heart Association (NYHA) Classification system categorizes heart failure based on functional limitations and symptoms:
NYHA Class | Symptoms | Functional Capacity | Nursing Considerations |
---|---|---|---|
Class I | No symptoms with ordinary activity | No limitation of physical activity | Focus on prevention, education, and monitoring |
Class II | Symptoms with ordinary activity | Slight limitation of physical activity | Symptom management, activity modification, diet counseling |
Class III | Symptoms with less than ordinary activity | Marked limitation of physical activity | Intensive medication management, activity planning, frequent monitoring |
Class IV | Symptoms at rest | Unable to perform any physical activity without discomfort | Comprehensive care, frequent assessment, palliative care considerations |
Classification by Ejection Fraction
HFrEF (Heart Failure with reduced Ejection Fraction)
- EF ≤ 40%
- Systolic dysfunction
- Decreased contractility
- Responds to medications that reduce afterload and enhance inotropy
HFpEF (Heart Failure with preserved Ejection Fraction)
- EF ≥ 50%
- Diastolic dysfunction
- Impaired relaxation and filling
- Focuses on managing volume status and controlling heart rate
HFmrEF (Heart Failure with mildly reduced Ejection Fraction)
- EF 41-49%
- Mixed systolic and diastolic dysfunction
- Treatment approaches similar to HFrEF
American College of Cardiology/American Heart Association Stages
Stage A
High risk for developing HF but without structural heart disease or symptoms
Examples: Hypertension, diabetes, obesity, metabolic syndrome
Stage B
Structural heart disease but without signs or symptoms of HF
Examples: Previous MI, LV hypertrophy, asymptomatic valve disease
Stage C
Structural heart disease with prior or current symptoms of HF
Examples: Shortness of breath, fatigue, reduced exercise tolerance
Stage D
Refractory HF requiring specialized interventions
Examples: Marked symptoms at rest despite optimal medical therapy
When documenting heart failure in nursing notes, be specific about the type of classification you’re using (NYHA, ACC/AHA, or EF-based) as each provides different but complementary information that guides treatment decisions. Remember that a patient may be Stage C in the ACC/AHA classification but NYHA Class I if their symptoms are well-controlled with medication.
The diagnosis of heart failure relies on a combination of clinical symptoms, physical examination findings, laboratory tests, and imaging studies. As a nurse, understanding these diagnostic criteria is crucial for early recognition and intervention.
Framingham Criteria for Heart Failure Diagnosis
The diagnosis requires the presence of 2 major criteria or 1 major and 2 minor criteria:
Major Criteria
- Paroxysmal nocturnal dyspnea
- Neck vein distension
- Pulmonary rales
- Cardiomegaly on chest X-ray
- Acute pulmonary edema
- S3 gallop
- Increased central venous pressure (>16 cm H₂O)
- Hepatojugular reflux
- Weight loss >4.5 kg in 5 days in response to treatment
Minor Criteria
- Bilateral ankle edema
- Nocturnal cough
- Dyspnea on ordinary exertion
- Hepatomegaly
- Pleural effusion
- Decrease in vital capacity by 1/3 from maximum
- Tachycardia (heart rate >120 beats/min)
Nursing Assessment
Heart Failure Assessment Mind Map
Key Diagnostic Tests
Diagnostic Test | Purpose | Findings in Heart Failure | Nursing Considerations |
---|---|---|---|
BNP/NT-proBNP | Detect ventricular wall stress | Elevated (BNP >100 pg/mL or NT-proBNP >300 pg/mL) | Obtain sample before treatment begins; explain purpose to patient |
Echocardiogram | Assess cardiac structure and function | Reduced EF, ventricular dilation, valve abnormalities | Explain procedure; no fasting required |
Chest X-ray | Evaluate pulmonary congestion | Cardiomegaly, pulmonary edema, pleural effusions | Remove metal objects; assist with positioning |
Electrocardiogram | Detect arrhythmias or ischemia | Left ventricular hypertrophy, bundle branch blocks, arrhythmias | Ensure proper lead placement; keep patient warm and comfortable |
Complete Blood Count | Screen for anemia or infection | Anemia may exacerbate or be caused by heart failure | Apply pressure after draw to prevent bruising |
Electrolytes | Monitor potassium and sodium | Hyponatremia common in advanced HF | Monitor for symptoms of electrolyte imbalance |
Renal Function | Assess kidney function | Elevated BUN/creatinine may indicate cardiorenal syndrome | Monitor I&O; assess for dehydration |
FAILURE: Mnemonic for Heart Failure Assessment
- F – Fluid status (edema, JVD, weight gain)
- A – Activity tolerance (NYHA class)
- I – Intake and output (fluid restriction compliance)
- L – Lung sounds (crackles, wheezing)
- U – Understanding of condition and medications
- R – Response to therapy (symptom improvement)
- E – Extremities (peripheral edema, capillary refill)
Nursing Assessment Tips
- Always compare current weight to dry/baseline weight
- Document edema using a standardized scale (e.g., 1+ to 4+)
- Auscultate lung bases thoroughly where crackles first appear
- Measure JVD with patient at 45° angle for consistency
- Ask about orthopnea in terms of number of pillows needed for sleep
Medication management is a cornerstone of heart failure treatment. Each drug class targets specific pathophysiological mechanisms to improve cardiac function, reduce symptoms, and increase survival.
Examples: Furosemide (Lasix), Bumetanide (Bumex), Torsemide (Demadex)
Mechanism: Inhibit sodium and water reabsorption in the nephron, promoting diuresis and reducing preload
Benefits: Reduce pulmonary congestion, peripheral edema, and dyspnea
Nursing Considerations:
- Monitor daily weights, I&O, and vital signs
- Assess for orthostatic hypotension before ambulation
- Monitor electrolytes (especially potassium, sodium, magnesium)
- Administer IV diuretics slowly to prevent ototoxicity
- Educate patients about the importance of timing doses (e.g., avoid taking at bedtime)
Examples: Lisinopril (Zestril), Enalapril (Vasotec), Ramipril (Altace)
Mechanism: Inhibit the conversion of angiotensin I to angiotensin II, reducing vasoconstriction and sodium retention
Benefits: Reduce afterload, improve cardiac output, slow ventricular remodeling, reduce mortality
Nursing Considerations:
- Monitor blood pressure closely (risk of hypotension, especially with first dose)
- Assess for dry cough (common side effect)
- Monitor renal function and potassium levels
- Educate patients to avoid NSAIDs, which can reduce effectiveness
- Contraindicated in pregnancy (teratogenic)
Examples: Losartan (Cozaar), Valsartan (Diovan), Candesartan (Atacand)
Mechanism: Block angiotensin II receptors directly, preventing vasoconstriction and aldosterone release
Benefits: Similar to ACE inhibitors but without cough side effect; alternative for ACE inhibitor-intolerant patients
Nursing Considerations:
- Monitor blood pressure and renal function
- Check for hyperkalemia
- Contraindicated in pregnancy
- Start at low dose and titrate according to response
Examples: Carvedilol (Coreg), Metoprolol succinate (Toprol XL), Bisoprolol (Zebeta)
Mechanism: Block beta-adrenergic receptors, reducing heart rate, contractility, and sympathetic overstimulation
Benefits: Reduce mortality, improve symptoms, prevent arrhythmias, and slow disease progression
Nursing Considerations:
- Start at low dose and titrate slowly (every 2 weeks)
- Monitor heart rate, blood pressure, and signs of worsening heart failure
- Assess for bradycardia, fatigue, dizziness, and bronchospasm
- Do not abruptly discontinue (risk of rebound hypertension or tachycardia)
- Educate patients that benefits may take weeks to months to appear
Examples: Spironolactone (Aldactone), Eplerenone (Inspra)
Mechanism: Block aldosterone receptors, reducing sodium retention and cardiac fibrosis
Benefits: Reduce mortality and hospitalizations, particularly in moderate to severe HF
Nursing Considerations:
- Monitor potassium levels closely (risk of hyperkalemia)
- Assess renal function before and during therapy
- Watch for gynecomastia with spironolactone (less common with eplerenone)
- Avoid concurrent use with potassium supplements unless specifically prescribed
Example: Digoxin (Lanoxin)
Mechanism: Inhibits Na⁺/K⁺-ATPase, increasing intracellular calcium and enhancing contractility
Benefits: Improves symptoms and reduces hospitalizations
Nursing Considerations:
- Monitor heart rate (target often 60-80 bpm)
- Check for signs of toxicity (visual changes, nausea, confusion)
- Monitor serum digoxin levels (therapeutic: 0.5-1.0 ng/mL)
- Assess electrolytes (hypokalemia increases toxicity risk)
- Numerous drug interactions – thorough medication reconciliation needed
Examples: Dapagliflozin (Farxiga), Empagliflozin (Jardiance)
Mechanism: Inhibit sodium-glucose cotransporter-2 in kidneys, increasing glucose excretion and natriuresis
Benefits: Reduce hospitalizations for HF and cardiovascular death, even in non-diabetic patients
Nursing Considerations:
- Monitor renal function
- Assess for genital mycotic infections
- Watch for signs of euglycemic DKA in diabetic patients
- Encourage adequate hydration
- Newer addition to HF therapy guidelines (2021)
When administering multiple heart failure medications, be mindful of the “triple whammy” effect of combining ACE inhibitors/ARBs, diuretics, and NSAIDs. This combination significantly increases the risk of acute kidney injury, especially in elderly patients. Always review medication lists carefully and educate patients about avoiding over-the-counter NSAIDs unless approved by their provider.
Medication Titration in Heart Failure
Many heart failure medications require gradual dose increases (titration) to reach target doses while minimizing side effects. The nurse plays a crucial role in this process:
Baseline Assessment
Document vital signs, weight, symptoms, and lab values before initiating therapy
Start Low
Begin with the lowest effective dose to minimize side effects
Monitor Response
Assess for therapeutic effects and side effects before each dose increase
Gradual Increases
Follow recommended titration schedules (usually every 2-4 weeks for beta-blockers and ACE inhibitors)
Reach Target Dose
Aim for evidence-based target doses or highest tolerated dose
Medication Safety Alert
Common heart failure medication interactions and concerns:
- NSAIDs can reduce the effectiveness of ACE inhibitors and diuretics, and worsen fluid retention
- Potassium supplements with ACE inhibitors or aldosterone antagonists increase hyperkalemia risk
- Calcium channel blockers (verapamil, diltiazem) may worsen heart failure and should generally be avoided
- Metformin should be used cautiously in HF due to lactic acidosis risk
- Beta-blockers can mask hypoglycemia symptoms in diabetic patients
Comprehensive nursing management of heart failure focuses on monitoring, symptom management, education, and emotional support. The nurse serves as the coordinator of care and patient advocate throughout the illness trajectory.
Key Areas of Heart Failure Nursing Management
Nursing Assessment and Monitoring
Assessment Parameter | What to Monitor | Frequency | Warning Signs |
---|---|---|---|
Vital Signs | BP, HR, RR, temperature, O2 saturation | Every 4 hours or more frequently during acute exacerbations | Hypotension, tachycardia, tachypnea, decreased SpO2 |
Weight | Daily weight, trend over time | Daily, same time, same clothes, after voiding | Gain of >2 lbs in 24 hours or >5 lbs in a week |
Fluid Status | Intake and output, edema, JVD | Every shift in acute care; daily in outpatient | Negative I/O balance, increasing edema, JVD >8 cm |
Respiratory Status | Breath sounds, work of breathing, orthopnea | Every 4 hours in acute care; each visit in outpatient | Crackles, wheezing, increased work of breathing |
Activity Tolerance | Ability to perform ADLs, exercise capacity | Daily in acute care; each visit in outpatient | Increased fatigue, decreased exercise capacity |
Medication Adherence | Understanding of regimen, side effects, barriers | Daily in acute care; each visit in outpatient | Confusion about medications, reported non-adherence |
Nutritional Status | Sodium intake, fluid restrictions, appetite | Daily in acute care; each visit in outpatient | Poor appetite, sodium/fluid restriction non-adherence |
Nursing Diagnoses and Interventions
Decreased Cardiac Output
Interventions:
- Monitor vital signs, especially heart rate and blood pressure
- Assess for signs of poor tissue perfusion (capillary refill, cognition)
- Administer medications as prescribed and monitor for effectiveness
- Position with head of bed elevated 30° to reduce cardiac workload
- Provide rest periods between activities
- Monitor oxygen saturation and administer oxygen as ordered
Excess Fluid Volume
Interventions:
- Monitor daily weights, intake and output, and edema
- Administer diuretics as prescribed and assess response
- Implement fluid restrictions as ordered
- Elevate edematous extremities when sitting or lying
- Auscultate lung sounds for crackles
- Educate on sodium restriction and fluid management
Activity Intolerance
Interventions:
- Assess activity tolerance using standardized tools
- Plan activities with scheduled rest periods
- Prioritize essential activities and assist as needed
- Implement progressive activity protocol as tolerated
- Monitor response to activity (vital signs, dyspnea, fatigue)
- Teach energy conservation techniques
Ineffective Breathing Pattern
Interventions:
- Position to optimize breathing (semi-Fowler’s or high Fowler’s)
- Administer oxygen therapy as prescribed
- Monitor respiratory rate, depth, and effort
- Teach pursed-lip breathing techniques
- Encourage deep breathing exercises
- Assess for and promptly report signs of respiratory distress
Deficient Knowledge
Interventions:
- Assess baseline knowledge of heart failure and self-care
- Provide education on disease process, medications, and warning signs
- Use teach-back method to verify understanding
- Provide written materials appropriate to literacy level
- Include family/caregivers in education sessions
- Reinforce teaching at each encounter
Risk for Impaired Skin Integrity
Interventions:
- Assess skin condition, especially in edematous areas
- Implement regular repositioning schedule
- Keep skin clean and dry, using mild cleansers
- Apply moisturizers to prevent dryness and cracking
- Use pressure-relieving devices as needed
- Monitor for early signs of skin breakdown
Patient Education
HEART: Heart Failure Education Mnemonic
- H – Home monitoring (daily weights, symptom tracking)
- E – Exercise and activity recommendations
- A – Adherence to medications and follow-up appointments
- R – Restrictions (sodium, fluid) and diet guidelines
- T – Treatment plan and when to call healthcare provider
Education Topic | Key Points to Teach | Teaching Strategies |
---|---|---|
Sodium Restriction |
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Medication Management |
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Symptom Recognition |
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Activity and Exercise |
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Weight Monitoring |
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When to Seek Medical Attention
Teach patients to contact their healthcare provider immediately if they experience:
- Weight gain of >2 lbs in 24 hours or >5 lbs in a week
- Increased swelling in legs, ankles, or abdomen
- Increased shortness of breath or difficulty breathing, especially when lying down
- Persistent cough or wheezing
- Increased fatigue or decreased ability to perform daily activities
- Dizziness or lightheadedness
- Rapid or irregular heartbeat
Discharge Planning and Transitional Care
Medication Reconciliation
Review all medications, ensure patient has access to them, and understanding of new regimen
Follow-up Appointments
Schedule follow-up within 7 days of discharge; ensure patient has transportation
Home Care Needs
Arrange home health services, medical equipment, and caregiver support as needed
Self-Care Education
Provide written instructions on medications, diet, activity, and symptom monitoring
Red Flags
Clear guidance on when to call provider vs. when to go to emergency department
Reducing Hospital Readmissions
Evidence-based strategies to prevent heart failure readmissions:
- Implement structured telephone follow-up within 48-72 hours of discharge
- Use teach-back method to confirm understanding of discharge instructions
- Schedule early post-discharge follow-up with provider (within 7 days)
- Refer to heart failure disease management programs when available
- Ensure seamless communication between hospital and community providers
- Provide patient-centered education that addresses individual barriers
- Consider palliative care consultation for advanced heart failure
Lifestyle modifications are fundamental to heart failure management and can significantly improve quality of life and reduce hospitalizations. Nurses play a crucial role in helping patients implement and maintain these changes.
Sodium Restriction
Most patients with heart failure benefit from limiting sodium intake to 2000-3000 mg/day.
Teaching Points for Sodium Restriction
- Read food labels (sodium content per serving)
- Avoid processed foods (canned soups, frozen meals, deli meats)
- Use herbs and spices instead of salt
- Choose fresh fruits and vegetables
- Be cautious with “salt substitutes” (may contain potassium)
- Limit restaurant dining or research low-sodium options
Fluid Management
Many patients with heart failure require fluid restrictions, typically 1.5-2 liters per day.
Teaching Points for Fluid Management
- Measure and track daily fluid intake
- Remember that foods like soup, ice cream, and gelatin count as fluids
- Distribute fluids throughout the day
- Use small cups to make portions seem larger
- Freeze allowed fluids as ice chips (takes longer to consume)
- Use sugar-free hard candies or gum to relieve dry mouth
Physical Activity and Exercise
NYHA Class | Exercise Recommendations | Precautions |
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Class I |
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Class II |
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Class III |
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Class IV |
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For heart failure patients beginning an exercise program, teach them to use the “talk test” as a simple way to gauge exercise intensity. Patients should be able to carry on a conversation while exercising. If they cannot speak in full sentences, they should reduce the intensity of their activity.
Weight Management
Weight management in heart failure has dual aspects: monitoring for fluid retention and maintaining a healthy BMI.
Daily Weight Monitoring
- Weigh daily, same time, same clothes, after voiding
- Use the same scale each time
- Record weight in a log
- Report gain of >2 lbs in 24 hours or >5 lbs in a week
- Understand the difference between fluid weight and true weight gain
Healthy BMI Goals
- Target BMI 18.5-24.9 kg/m²
- Cardiac cachexia is a poor prognostic sign
- Weight loss should be gradual (1-2 lbs/week)
- Focus on nutrient-dense, heart-healthy foods
- Collaborate with dietitian for individualized plan
Additional Lifestyle Considerations
Smoking Cessation
- Assess readiness to quit using stages of change model
- Discuss pharmacological aids (nicotine replacement, bupropion, varenicline)
- Refer to smoking cessation programs
- Provide resources for quitlines and support groups
- Follow up on progress at each visit
Alcohol Limitation
- For non-alcoholic cardiomyopathy: limit to 1 drink or less per day
- For alcoholic cardiomyopathy: complete abstinence recommended
- Educate about alcohol’s negative effects on heart function
- Screen for alcohol use disorder and refer as needed
- Discuss alcohol’s interaction with medications
Sleep Management
- Screen for sleep-disordered breathing (common in HF)
- Elevate head of bed 30° to reduce orthopnea
- Recommend avoiding fluids before bedtime
- Establish regular sleep schedule
- Discuss proper CPAP use if prescribed
Stress Management
- Teach relaxation techniques (deep breathing, meditation)
- Encourage social support and connection
- Consider referral for cognitive behavioral therapy
- Screen for depression and anxiety
- Explore mindfulness-based stress reduction programs
Travel Considerations for Heart Failure Patients
When counseling heart failure patients about travel, address these key points:
- Consult with healthcare provider before planning extended travel
- Carry a current medication list and adequate supply (plus extra)
- Consider medical alert identification
- Research medical facilities at destination
- For air travel, request wheelchair assistance if needed
- Stay hydrated but maintain fluid restrictions
- Move frequently on long trips to prevent blood clots
- Consider travel insurance with medical coverage
Heart Failure Comprehensive Management Mind Map
Key Takeaways
- Pathophysiology: Heart failure involves decreased cardiac output, compensatory mechanisms that become maladaptive, and neurohormonal activation
- Classification: HF is categorized by affected side (left, right, biventricular), ejection fraction (HFrEF, HFmrEF, HFpEF), and functional capacity (NYHA Classes I-IV)
- Diagnosis: Based on clinical presentation, physical exam findings, and diagnostic tests (BNP, echocardiogram, chest X-ray)
- Treatment: Comprehensive approach including medications (diuretics, ACE inhibitors, beta-blockers), lifestyle modifications, and device therapy when indicated
- Nursing Management: Focuses on monitoring symptoms, administering medications, providing education, and supporting lifestyle modifications
ABCDES of Heart Failure Management
- A – Assessment (comprehensive and ongoing)
- B – Beta-blockers and other medications
- C – Cardiac rehabilitation and lifestyle changes
- D – Dietary modifications (sodium restriction)
- E – Education (patient and family)
- S – Self-monitoring (weight, symptoms)
Heart failure management is a marathon, not a sprint. The most successful nurses build therapeutic relationships with their heart failure patients, celebrating small victories and providing support during setbacks. Remember that improving quality of life is just as important as extending it. Your role in education, symptom management, and emotional support can make a profound difference in these patients’ lives.
- American Heart Association. (2023). Classes and Stages of Heart Failure. https://www.heart.org/en/health-topics/heart-failure/what-is-heart-failure/classes-of-heart-failure
- Inamdar, A. A., & Inamdar, A. C. (2025). Heart Failure (Congestive Heart Failure). StatPearls Publishing. https://www.ncbi.nlm.nih.gov/books/NBK430873/
- Mayo Clinic. (2025). Heart failure – Diagnosis and treatment. https://www.mayoclinic.org/diseases-conditions/heart-failure/diagnosis-treatment/drc-20373148
- Cleveland Clinic. (2024). Congestive Heart Failure: Symptoms, Stages & Treatment. https://my.clevelandclinic.org/health/diseases/17069-heart-failure-understanding-heart-failure
- Drazner, M. H. (2022). Heart failure: Clinical manifestations and diagnosis in adults. UpToDate. https://www.uptodate.com/contents/heart-failure-clinical-manifestations-and-diagnosis-in-adults
- Cole, J., & Florea, V. G. (2023). Pathophysiology of heart failure. PubMed Central. https://pmc.ncbi.nlm.nih.gov/articles/PMC7944197/
- American Heart Association. (2025). Medications Used to Treat Heart Failure. https://www.heart.org/en/health-topics/heart-failure/treatment-options-for-heart-failure/medications-used-to-treat-heart-failure
- Delaney, C., & Apostolidis, B. (2019). Medications for heart failure management: What nurses need to know. American Nurse. https://www.myamericannurse.com/medications-heart-failure-management/
- Tanna, M. S., & Mozolevska, V. (2023). Heart Failure (Congestive Heart Failure) (Nursing). StatPearls Publishing. https://www.ncbi.nlm.nih.gov/books/NBK574497/
- Merck Manual. (2024). New York Heart Association (NYHA) Classification of Heart Failure. https://www.merckmanuals.com/professional/multimedia/table/new-york-heart-association-nyha-classification-of-heart-failure